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Paper Published

Mediators of Inflammation Volume 2015 (2015), Article ID 176983, 10 pages Loss of AMP-Activated Protein Kinase Induces Mitochondrial Dysfunction and Proinflammatory Response in Unstimulated Abcd1-Knockout Mice Mixed Glial Cells.

Published OnlineFirst on May 10, 2013; DOI: 10.1158/1541-7786.MCR-12-0686. Downloaded from mcr.aacrjournals.org on August 7, 2015. 2013 American Association for Cancer Research microRNA-155 deficiency in bone marrow results in enhanced tumor metastasis.

PLoS ONE (Impact Factor: 3.53). 07/2013; 8(7):e70712. DOI: 10.1371/journal.pone.0070712 Aug 07, 2015; Histone Deacetylase Inhibitor Upregulates Peroxisomal Fatty Acid Oxidation and Inhibits Apoptotic Cell Death in Abcd1-Deficient Glial Cells.

Biochemical and Biophysical Research Communications Volume 445, Issue 1, 28 February 2014, Pages 126–131 Loss of AMP-activated protein kinase in X-linked adrenoleukodystrophy patient-derived fibroblasts and lymphocytes.

Cancer Res November 1, (2014) 74:6048-6059; Reducing CD73 Expression by IL1β-Programmed Th17 Cells Improves Immunotherapeutic Control of Tumors.

 

Breast Cancer Research and Treatment November (2014), Volume 148, Issue 2, pp 291-302 Emodin suppresses pulmonary metastasis of breast cancer accompanied with decreased macrophage recruitment and M2 polarization in the lungs.

 

PLoS ONE 2012, 7(1): e29830.  ONE Retinal Muller Glia Initiate Innate Response to Infectious Stimuli via Toll-Like Receptor Signaling Ashok Kumar, Nazeem Shamsuddin.

 

J Lipid Res 2011 Nov; 52(11): 2056-69. HDAC inhibitor SAHA normalizes the levels of VLCFAs in human skin fibroblasts from X-ALD patients and downregulates the expression of proinflammatory cytokines in Abcd1/2-silenced mouse astrocytes.

 

J Immunol. 2011 Jun 15; 186(12): 7089-97. TLR2 mediates the innate response of retinal Muller glia to Staphylococcus aureus.

 

Biochimica et Biophysica Acta 1831 (2013) 747–758. Caffeic acid phenethyl ester induces adrenoleukodystrophy (Abcd2) gene in human X-ALD fibroblasts and inhibits the proinflammatory

response in Abcd1/2 silenced mouse primary astrocytes.

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